Preliminary studies show conolidine might inhibit distinct ion channels, cutting down neuronal excitability and restricting ache signals. This mechanism is especially relevant in neuropathic soreness, wherever abnormal signaling causes persistent distress. Also, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to soreness notion. Computational docking exp... https://pikm627zau4.wikifordummies.com/user